Staphylococcus aureus colonies can unfold on soft agar plates. We when compared colony spreading of clinically isolated methicillin-delicate S.
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Preceding studies have indicated that the silkworm design is useful for pinpointing virulence genes of Staphylococcus aureus, a human pathogenic bacterium.
Sugar-responsive gene expression and also the agr process are needed for colony spreading in Staphylococcus aureus
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cell area and found that δ-toxin inhibited colony-spreading exercise by reducing the quantities of PSMα1–4 within the cell surface area. The findings with the present study discovered a novel purpose of mobile surface PSMs to modulate S
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We previously reported which the cvfB gene (SA1223) of Staphylococcus aureus is liable for the virulence of the pathogenic bacterium.